High purity custom peptide MOTS-c / MOTS3 / mots peptide from China CAS NO.1627580-64-6

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Min.Order: 100 Milligram
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Product Details

Keywords

mots peptide
MOTS-c
MOTS3

Quick Details

ProName: High purity custom peptide MOTS-c / ...
CasNo: 1627580-64-6
Molecular Formula: C???H???N??O??S?
Appearance: White powder
Application: PEPTIDE
DeliveryTime: 3-5 working days after synthsized
PackAge: 1gram per bottle(plastic bottle or gla...
Port: Chengdu
ProductionCapacity: 1000 Gram/Month
Purity: 95%MIN
Storage: 1000g/Month
Transportation: Normal Temperature
LimitNum: 100 Milligram
Moisture Content: N/A
Impurity: N/A
CAS: 1627580-64-6
Color: white
shelf life: 2 years
brand: youngshe
Transportation:: DHL, FeDex and EMS

Superiority

Chengdu YoungShe Chemical Co.,Ltd is a dynamic and progressive cosmetic peptides supplier in China. We are dedicating to be a professional, efficient,and reliable partner for global customers on cosmetic peptides. You can select more than 170 kinds of raw cosmetic peptide ingredients here.And we are keeping developing new products and continuously marketing them for sale.Youngshe Chem provide a one-stop service for raw cosmetic peptide ingredients.You will save lots of time ,energy, money and have a very pleasant experience with us.Since Dec,2014,YoungShe group has expanded their business to botanical cosmetic active ingredients and related.

Details

MOTS-c

Name: MOTS-c

CAS No. : 1627580-64-6

Sequence:H-Met-Arg-Trp-Gln-Glu-Met-Gly-Tyr-Ile-Phe-Tyr-Pro-Arg-Lys-Leu-Arg-OH

Molecular formula: C???H???N??O??S?

Molecular weight : 2174.6

Purity: 95 %

Source: synthetic

MSDS and COA: available

Mitochondria are known to be functional organelles, but their role as a signaling unit is increasingly being appreciated. The identification of a short open reading frame (sORF) in the mitochondrial DNA (mtDNA) that encodes a signaling peptide, humanin, suggests the possible existence of additional sORFs in the mtDNA. Here we report a sORF within the mitochondrial 12S rRNA encoding a 16-amino-acid peptide named MOTS-c (mitochondrial open reading frame of the 12S rRNA-c) that regulates insulin sensitivity and metabolic homeostasis. Its primary target organ appears to be the skeletal muscle, and its cellular actions inhibit the folate cycle and its tethered de novo purine biosynthesis, leading to AMPK activation. MOTS-c treatment in mice prevented age-dependent and high-fat-diet-induced insulin resistance, as well as diet-induced obesity. These results suggest that mitochondria may actively regulate metabolic homeostasis at the cellular and organismal level via peptides encoded within their genome.

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